Cilia is an open access peer-reviewed journal that publishes high quality basic and translational research on the biology of cilia and diseases associated with ciliary dysfunction. Research approaches include cell and developmental biology, use of model organisms, and human and molecular genetics.
Editors-in-Chief
- Philip L Beales, University College London
- Peter K Jackson, Genentech Inc
Articles
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Review
Cilia 2013, 2:6 (29 April 2013)Functional aspects of primary cilia in signaling, cell cycle and tumorigenesis
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Research
Cilia 2013, 2:5 (25 March 2013)The effect of ethanol and acetaldehyde on brain ependymal and respiratory ciliary beat frequency
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Review
Cilia 2013, 2:3 (5 February 2013)Pancreatic cyst development: insights from von Hippel-Lindau disease
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Research
Cilia 2013, 2:2 (31 January 2013)Reduced cilia frequencies in human renal cell carcinomas versus neighboring parenchymal tissue
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Useful links
Editors' profiles
Philip Beales
Philip Beales is Professor of Medical Genetics at the Institute of Child Health, University College London, and a consultant clinical geneticist at Guy’s Hospital and Great Ormond Street Hospital in London. His research is focussed on the study of the role of primary cilia in disease and development. He has a longstanding interest in the ciliopathy Bardet-Biedl syndrome, and his laboratory was instrumental in unveiling the role of primary cilia dysfunction in the pathogenesis of this rare congenital condition.
Peter Jackson
Peter Jackson is a Staff Scientist at Genentech Inc. in San Francisco, California, where he is involved in defining the strategy for development of therapeutics for critical physiological pathways including tumour metabolism and the cell cycle. Before joining Genentech in 2005, he spent 10 years on the faculty at Stanford University School of Medicine. At Genentech, his laboratory has been involved in studies of cell cycle biochemistry, particularly the role of the ubiquitin-proteasome system in cell cycle regulation, with a central finding being the discovery of E3 ubiquitin ligase inhibitors.
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